
Cover image: Human dorsal root ganglia xenotransplanted into a mouse model and infected with varicella-zoster virus (VZV). VZV, the agent of varicella (chickenpox), remains latent in dorsal root ganglia and may reactivate later in life to cause herpes zoster (shingles). The VZV gene product glycoprotein I is essential for the transition to viral persistence in vivo. See the article by Zerboni et al. on pages 14086–14091. Photograph courtesy of Leigh Zerboni.